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Adaptive Mutations in the V3 Loop of gp120 Enhance Fusogenicity of Human Immunodeficiency Virus Type 1 and Enable Use of a CCR5 Coreceptor That Lacks the Amino-Terminal Sulfated Region

To identify sites in gp120 that interact with the CCR5 coreceptor and to analyze the mechanisms of infection, we selected variants of the CCR5-dependent JRCSF molecular clone of human immunodeficiency virus type 1 (HIV-1) that adapted to replicate in HeLa-CD4 cells that express the mutant coreceptor...

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Udgivet i:J Virol
Principais autores: Platt, Emily J., Kuhmann, Shawn E., Rose, Patrick P., Kabat, David
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Microbiology (ASM) 2001
Fag:
Virus-Cell Interactions
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC116123/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11711617/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.75.24.12266-12278.2001
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