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Modulation of transmembrane signalling in HL-60 granulocytes by tumour necrosis factor-alpha.

Differentiated HL-60 granulocytes were used to study the mechanism by which tumour necrosis factor-alpha (TNF) enhances responses to N-formyl-methionyl-leucylphenylalanine (FMLP). Cultivation of differentiated HL-60 cells with 100 units of TNF/ml for 24 h resulted in a 3-fold increase in superoxide...

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Autors principals: McLeish, K R, Klein, J B, Schepers, T, Sonnenfeld, G
Format: Artigo
Idioma:Inglês
Publicat: 1991
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1151626/
https://ncbi.nlm.nih.gov/pubmed/1659380
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