Okadaic acid identifies a phosphorylation/dephosphorylation cycle controlling the inhibitory guanine-nucleotide-binding regulatory protein Gi2.

Παρόμοια τεκμήρια

• Multi-site phosphorylation of the inhibitory guanine nucleotide regulatory protein Gi-2 occurs in intact rat hepatocytes.
  ανά: Morris, N J, κ.ά.
  Έκδοση: (1994)
• Diabetes-induced alterations in the expression, functioning and phosphorylation state of the inhibitory guanine nucleotide regulatory protein Gi-2 in hepatocytes.
  ανά: Bushfield, M, κ.ά.
  Έκδοση: (1990)
• Diabetes abolishes the GTP-dependent, but not the receptor-dependent inhibitory function of the inhibitory guanine-nucleotide-binding regulatory protein (Gi) on adipocyte adenylate cyclase activity.
  ανά: Strassheim, D, κ.ά.
  Έκδοση: (1990)
• Demonstration of inhibitory guanine nucleotide regulatory protein (Gi) function in liver and hepatocyte membranes from streptozotocin-treated rats.
  ανά: Kirkham, D M, κ.ά.
  Έκδοση: (1992)
• Treatment of streptozotocin-diabetic rats with metformin restores the ability of insulin to inhibit adenylate cyclase activity and demonstrates that insulin does not exert this action through the inhibitory guanine nucleotide regulatory protein Gi.
  ανά: Gawler, D, κ.ά.
  Έκδοση: (1988)