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Defective lysosomal release of glycoprotein-derived sialic acid in fibroblasts from patients with sialic acid storage disease.

Fibroblasts from patients with sialic acid storage disease (SASD), sialidosis, mucolipidosis II, and from normal controls, were incubated in the presence of the glycoprotein fetuin that was tritium-labelled in its sialic acid residues by the periodate/[3H]borohydride reduction method, and the fate o...

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Bibliographische Detailangaben
Hauptverfasser: Mendla, K, Baumkötter, J, Rosenau, C, Ulrich-Bott, B, Cantz, M
Format: Artigo
Sprache:Inglês
Veröffentlicht: 1988
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1148842/
https://ncbi.nlm.nih.gov/pubmed/2451509
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