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Defective lysosomal release of glycoprotein-derived sialic acid in fibroblasts from patients with sialic acid storage disease.

Fibroblasts from patients with sialic acid storage disease (SASD), sialidosis, mucolipidosis II, and from normal controls, were incubated in the presence of the glycoprotein fetuin that was tritium-labelled in its sialic acid residues by the periodate/[3H]borohydride reduction method, and the fate o...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Mendla, K, Baumkötter, J, Rosenau, C, Ulrich-Bott, B, Cantz, M
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1988
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC1148842/
https://ncbi.nlm.nih.gov/pubmed/2451509
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