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Fibrin cross-linking in congenital factor XIII deficiency.

Homozygous patients with factor XIII deficiency are devoid of immunologically identifiable A protein, the active enzymatic component. Quantitative studies of transamidase activity of the factor are available in only a few cases, and the fibrin cross-linking pattern is not well known. The present pap...

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Détails bibliographiques
Auteurs principaux: Rodeghiero, F, Barbui, T
Format: Artigo
Langue:Inglês
Publié: 1980
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC1146106/
https://ncbi.nlm.nih.gov/pubmed/7400341
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