Lipopeptides activate Gi-proteins in dibutyryl cyclic AMP-differentiated HL-60 cells.

Lignende værker

• Mastoparan may activate GTP hydrolysis by Gi-proteins in HL-60 membranes indirectly through interaction with nucleoside diphosphate kinase.
  af: Klinker, J F, et al.
  Udgivet: (1994)
• Transcriptional inactivation of c-myc and the transferrin receptor in dibutyryl cyclic AMP-treated HL-60 cells.
  af: Trepel, J B, et al.
  Udgivet: (1987)
• Differential regulation of amounts of the guanine-nucleotide-binding proteins Gi and Go in neuroblastoma x glioma hybrid cells in response to dibutyryl cyclic AMP.
  af: Mullaney, I, et al.
  Udgivet: (1988)
• The inhibitory effect of dibutyryl cyclic AMP on docosahexaenoic acid-induced apoptosis in HL-60 cells through activation of the phosphatidylinositol-3 kinase pathway
  af: Miura, Yoshie, et al.
  Udgivet: (2005)
• Formyl peptides and ATP stimulate Ca2+ and Na+ inward currents through non-selective cation channels via G-proteins in dibutyryl cyclic AMP-differentiated HL-60 cells. Involvement of Ca2+ and Na+ in the activation of beta-glucuronidase release and superoxide production.
  af: Krautwurst, D, et al.
  Udgivet: (1992)