טוען...
Amino Acid 36 in the Human Immunodeficiency Virus Type 1 gp41 Ectodomain Controls Fusogenic Activity: Implications for the Molecular Mechanism of Viral Escape from a Fusion Inhibitor
We have previously described a human immunodeficiency virus type 1 (HIV-1) proviral clone, pL2, derived from defective viral particles with higher fusogenicity than the prototypic NL4-3 virus. In this study, we attempted to determine the region that confers the enhanced fusion activity by creating e...
שמור ב:
| Main Authors: | , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Microbiology
2005
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| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1091722/ https://ncbi.nlm.nih.gov/pubmed/15857986 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.79.10.5996-6004.2005 |
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