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Secreted Aspartyl Proteinases and Interactions of Candida albicans with Human Endothelial Cells

The endothelial cell interactions of homozygous null mutants of Candida albicans that were deficient in secreted aspartyl proteinase 1 (Sap1), Sap2, or Sap3 were investigated. Only Sap2 was found to contribute to the ability of C. albicans to damage endothelial cells and stimulate them to express E-...

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Vydáno v:Infect Immun
Hlavní autoři: Ibrahim, Ashraf S., Filler, Scott G., Sanglard, Dominique, Edwards, John E., Hube, Bernhard
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Microbiology (ASM) 1998
Témata:
On-line přístup:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC108304/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9596782/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.66.6.3003-3005.1998
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