The Putative Oncoprotein Bcl-3 Induces Cyclin D1 To Stimulate G(1) Transition

Bcl-3 is a distinctive member of the IκB family of NF-κB inhibitors because it can function to coactivate transcription. A potential involvement of Bcl-3 in oncogenesis is highlighted by the fact that it was cloned due to its location at a breakpoint junction in some cases of human B-cell chronic ly...

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Autores principales: Westerheide, Sandy D., Mayo, Marty W., Anest, Vasiliki, Hanson, Julie L., Baldwin, Albert S.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Microbiology 2001
Materias:
Cell Growth and Development
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC100006/
https://ncbi.nlm.nih.gov/pubmed/11713278
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.21.24.8428-8436.2001
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