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The Putative Oncoprotein Bcl-3 Induces Cyclin D1 To Stimulate G(1) Transition
Bcl-3 is a distinctive member of the IκB family of NF-κB inhibitors because it can function to coactivate transcription. A potential involvement of Bcl-3 in oncogenesis is highlighted by the fact that it was cloned due to its location at a breakpoint junction in some cases of human B-cell chronic ly...
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| Autori principali: | , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Microbiology
2001
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC100006/ https://ncbi.nlm.nih.gov/pubmed/11713278 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.21.24.8428-8436.2001 |
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