SLC35A2 Deficiency Promotes an Epithelial-to-Mesenchymal Transition-like Phenotype in Madin–Darby Canine Kidney Cells

In mammalian cells, SLC35A2 delivers UDP–galactose for galactosylation reactions that take place predominantly in the Golgi lumen. Mutations in the corresponding gene cause a subtype of a congenital disorder of glycosylation (SLC35A2-CDG). Although more and more patients are diagnosed with SLC35A2-C...

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Egile Nagusiak:	Magdalena Kot, Ewa Mazurkiewicz, Maciej Wiktor, Wojciech Wiertelak, Antonina Joanna Mazur, Andrei Rahalevich, Mariusz Olczak, Dorota Maszczak-Seneczko
Formatua:	Artigo
Hizkuntza:	Inglês
Argitaratua:	MDPI AG 2022-07-01
Saila:	Cells
Gaiak:	solute carrier family 35 member A2 (SLC35A2) Madin–Darby canine kidney (MDCK) cells epithelial-to-mesenchymal transition (EMT) UDP-galactose glycosylation Golgi complex
Sarrera elektronikoa:	https://www.mdpi.com/2073-4409/11/15/2273
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