FXR acts as a therapeutic target for ulcerative colitis via suppressing ferroptosis
Abstract Background The involvement of ferroptosis in ulcerative colitis (UC) is increasingly acknowledged. Several investigations have revealed the various mechanisms by which the farnesoid X receptor (FXR) inhibits ferroptosis in certain diseases; however, its potential modulation of ferroptosis i...
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| Hlavní autoři: | , , , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
BMC
2025-07-01
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| Edice: | Molecular Medicine |
| Témata: | |
| On-line přístup: | https://doi.org/10.1186/s10020-025-01305-3 |
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