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The Interaction between SARS-CoV-2 Nucleocapsid Protein and UBC9 Inhibits MAVS Ubiquitination by Enhancing Its SUMOylation

Severe COVID-19 patients exhibit impaired IFN-I response due to decreased IFN-β production, allowing persistent viral load and exacerbated inflammation. While the SARS-CoV-2 nucleocapsid (N) protein has been implicated in inhibiting innate immunity by interfering with IFN-β signaling, the specific u...

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Autori principali: Congcong Huang, Yiping Yin, Pan Pan, Yanping Huang, Siwei Chen, Junkai Chen, Ju Wang, Guoqing Xu, Xuan Tao, Xiao Xiao, Jian Li, Jing Yang, Zhixiong Jin, Bei Li, Zhaohui Tong, Weixing Du, Long Liu, Zhixin Liu
Natura: Artigo
Lingua:Inglês
Pubblicazione: MDPI AG 2023-11-01
Serie:Viruses
Soggetti:
SARS-CoV-2
nucleocapsid protein
UBC9
MAVS
SUMOylation
ubiquitination
Accesso online:https://www.mdpi.com/1999-4915/15/12/2304
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