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NMDA receptors mediate synaptic depression, but not spine loss in the dentate gyrus of adult amyloid Beta (Aβ) overexpressing mice

Abstract Amyloid beta (Aβ)-mediated synapse dysfunction and spine loss are considered to be early events in Alzheimer’s disease (AD) pathogenesis. N-methyl-D-aspartate receptors (NMDARs) have previously been suggested to play a role for Amyloid beta (Aβ) toxicity. Pharmacological block of NMDAR subu...

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Principais autores: Michaela Kerstin Müller, Eric Jacobi, Kenji Sakimura, Roberto Malinow, Jakob von Engelhardt
פורמט: Artigo
שפה:Inglês
יצא לאור: BMC 2018-10-01
סדרה:Acta Neuropathologica Communications
נושאים:
NMDA receptor
Amyloid Beta
Alzheimer’s disease
GluN2B
GluN2A
גישה מקוונת:http://link.springer.com/article/10.1186/s40478-018-0611-4
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