RYBP promotes HIV-1 latency through promoting H2AK119ub and decreasing H3K4me3

Abstract Background Acquired immunodeficiency syndrome (AIDS) cannot be completely cured, and the main obstacle is the existence of viral reservoirs. However, we currently do not fully understand the molecular mechanisms by which HIV-1 latency is established and maintained. Methods Here, based on en...

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Principais autores:	Xinyi Yang, Yuqi Zhu, Xiaying Zhao, Jingna Xun, Xingyu Wang, Yipeng Cheng, Su Xiong, Xingwen Yu, Suixiang Li, Danqing Wang, Zhiliang Hu, Yinzhong Shen, Shibo Jiang, Hongzhou Lu, Gang Wang, Huanzhang Zhu
格式:	Artigo
語言:	Inglês
出版:	BMC 2025-05-01
叢編:	Cell Communication and Signaling
主題:	RYBP KDM2B HIV-1 latency H2AK119ub EnChIP
在線閱讀:	https://doi.org/10.1186/s12964-025-02221-z
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