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Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

<p>Abstract</p> <p>Background</p> <p>Neoplastic transformation of cultured cells by a number of oncogenes such as <it>src</it> suppresses gap junctional, intercellular communication (GJIC); however, the role of Src and its effector Signal transducer and activator of transcription-3 (Stat3) upon GJIC...

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Principais autores: Geletu Mulu, Arulanandam Rozanne, Greer Samantha, Trotman-Grant Aaron, Tomai Evangelia, Raptis Leda
Format: Artigo
Sprog:Inglês
Udgivet: BMC 2012-12-01
Serier:BMC Cancer
Fag:
Stat3
Electroporation
Indium-Tin oxide
Gap junctions
Src
Cell to cell adhesion
Lung cancer
Online adgang:http://www.biomedcentral.com/1471-2407/12/605
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