Stat3 is a positive regulator of gap junctional intercellular communication in cultured, human lung carcinoma cells

<p>Abstract</p> <p>Background</p> <p>Neoplastic transformation of cultured cells by a number of oncogenes such as <it>src</it> suppresses gap junctional, intercellular communication (GJIC); however, the role of Src and its effector Signal transducer and activator of transcription-3 (Stat3) upon GJIC...

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Autori principali:	Geletu Mulu, Arulanandam Rozanne, Greer Samantha, Trotman-Grant Aaron, Tomai Evangelia, Raptis Leda
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	BMC 2012-12-01
Serie:	BMC Cancer
Soggetti:	Stat3 Electroporation Indium-Tin oxide Gap junctions Src Cell to cell adhesion Lung cancer
Accesso online:	http://www.biomedcentral.com/1471-2407/12/605
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