Glucose-dependent insulinotropic peptide receptor overexpression in adrenocortical hyperplasia in MEN1 syndrome without loss of heterozygosity at the 11q13 locus

BACKGROUND: The molecular mechanisms involved in the genesis of the adrenocortical lesions seen in MEN1 syndrome (ACL-MEN1) remain poorly understood; loss of heterozygosity at 11q13 and somatic mutations of MEN1 are not usually found in these lesions. Thus, additional genes must be involved in MEN1...

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Autors principals:	Marcia Helena Soares Costa, Sorahia Domenice, Rodrigo Almeida Toledo, Delmar Muniz L. Junior, Ana Claudia Latronico, Emilia Modolo Pinto, Sergio Pereira Almeida Toledo, Berenice Bilharinho Mendonca, Maria Candida Barisson Villares Fragoso
Format:	Artigo
Idioma:	Inglês
Publicat:	Elsevier España 2011-01-01
Col·lecció:	Clinics
Matèries:	MEN1 adrenocortical hyperplasia GIPR expression
Accés en línia:	http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1807-59322011000400002
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