Hyper-Acetylation of Histone H3K56 Limits Break-Induced Replication by Inhibiting Extensive Repair Synthesis.
Break-induced replication (BIR) has been implicated in restoring eroded telomeres and collapsed replication forks via single-ended invasion and extensive DNA synthesis on the recipient chromosome. Unlike other recombination subtypes, DNA synthesis in BIR likely relies heavily on mechanisms enabling...
שמור ב:
| Principais autores: | , , , , , |
|---|---|
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Public Library of Science (PLoS)
2015-02-01
|
| סדרה: | PLoS Genetics |
| גישה מקוונת: | https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1004990&type=printable |
| תגים: |
אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!
|
