Amyloid β induces interneuron-specific changes in the hippocampus of APPNL-F mice.

Alzheimer's disease (AD) is a neurodegenerative disorder characterized by cognitive decline and amyloid-beta (Aβ) depositions generated by the proteolysis of amyloid precursor protein (APP) in the brain. In APPNL-F mice, APP gene was humanized and contains two familial AD mutations, and APP-unlike o...

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Hlavní autoři:	Katalin E Sos, Márton I Mayer, Virág T Takács, Abel Major, Zsuzsanna Bardóczi, Barnabas M Beres, Tamás Szeles, Takashi Saito, Takaomi C Saido, István Mody, Tamás F Freund, Gábor Nyiri
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Public Library of Science (PLoS) 2020-01-01
Edice:	PLoS ONE
On-line přístup:	https://doi.org/10.1371/journal.pone.0233700
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