Monoammonium glycyrrhizinate ameliorates mitochondrial dysfunction-mediated oxidative stress and neuroinflammation via the NRF2/NQO1 axis after spinal cord injury

Background: Spinal cord injury (SCI)-induced mitochondrial dysfunction in microglia exacerbates neuroinflammation and neurological deficits. Monoammonium glycyrrhizinate (MAG), a bioactive liquorice-derived compound, exhibits anti-inflammatory and antioxidant properties; however, its effects on micr...

Täydet tiedot

Bibliografiset tiedot

Päätekijät:	Tianyi Wang, Jiale Huang, Jian Zhou, Mingjie Xia, Zheng Zhou, Qianqiu Li, Guanhua Xu, Zhanyang Qian, Zhiming Cui
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	Taylor & Francis Group 2025-12-01
Sarja:	Redox Report
Aiheet:	Spinal cord injury monoammonium glycyrrhizinate mitochondrial dysfunction neuroinflammation oxidative stress reactive oxygen species
Linkit:	https://www.tandfonline.com/doi/10.1080/13510002.2025.2585221
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