Truncation in the <it>tcdC </it>region of the <it>Clostridium difficile </it>PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A
<p>Abstract</p> <p>Background</p> <p>The increased severity of disease associated with the NAP1 strain of <it>Clostridium difficile </it>has been attributed to mutations to the <it>tcdC </it>gene which codes for a negative regulator of toxin production. To assess the role of hyper-production of Toxi...
محفوظ في:
| المؤلفون الرئيسيون: | , , , , |
|---|---|
| التنسيق: | Artigo |
| اللغة: | Inglês |
| منشور في: |
BMC
2009-06-01
|
| سلاسل: | BMC Infectious Diseases |
| الوصول للمادة أونلاين: | http://www.biomedcentral.com/1471-2334/9/103 |
| الوسوم: |
لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
|