Truncation in the <it>tcdC </it>region of the <it>Clostridium difficile </it>PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A
<p>Abstract</p> <p>Background</p> <p>The increased severity of disease associated with the NAP1 strain of <it>Clostridium difficile </it>has been attributed to mutations to the <it>tcdC </it>gene which codes for a negative regulator of toxin production. To assess the role of hyper-production of Toxi...
Kaydedildi:
| Asıl Yazarlar: | , , , , |
|---|---|
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
BMC
2009-06-01
|
| Seri Bilgileri: | BMC Infectious Diseases |
| Online Erişim: | http://www.biomedcentral.com/1471-2334/9/103 |
| Etiketler: |
Etiket eklenmemiş, İlk siz ekleyin!
|
