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Truncation in the <it>tcdC </it>region of the <it>Clostridium difficile </it>PathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A

<p>Abstract</p> <p>Background</p> <p>The increased severity of disease associated with the NAP1 strain of <it>Clostridium difficile </it>has been attributed to mutations to the <it>tcdC </it>gene which codes for a negative regulator of toxin production. To assess the role of hyper-production of Toxi...

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Detaylı Bibliyografya
Asıl Yazarlar: Levett Paul N, Boyd Dave, Murray Ruth, Mulvey Michael R, Alfa Michelle J
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: BMC 2009-06-01
Seri Bilgileri:BMC Infectious Diseases
Online Erişim:http://www.biomedcentral.com/1471-2334/9/103
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