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Lipid Nanoparticle‐Mediated Delivery of CRISPR‐Cas9 Against Rubicon Ameliorates NAFLD by Modulating CD36 Along with Glycerophospholipid Metabolism

Abstract Non‐alcoholic fatty liver disease (NAFLD) is a prominent cause of various chronic metabolic hepatic diseases with limited therapeutics. Rubicon, an essential regulator in lysosomal degradation, is reported to exacerbate hepatic steatosis in NAFLD mice and patients, indicating its probabilit...

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Principais autores: Yu Bai, Yanyang Nan, Tao Wu, An Zhu, Xinlei Xie, Yun Sun, Yong Deng, Zihan Dou, Xiaozhi Hu, Rongrui Zhou, Shuwen Xu, Yuanzhen Zhang, Jiajun Fan, Dianwen Ju
Formato: Artigo
Idioma:Inglês
Publicado em: Wiley 2024-08-01
coleção:Advanced Science
Assuntos:
CD36
CRISPR‐Cas9
glycerophospholipid metabolism
LNP
NAFLD
Rubicon
Acesso em linha:https://doi.org/10.1002/advs.202400493
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