RAGE mediates airway inflammation via the HDAC1 pathway in a toluene diisocyanate-induced murine asthma model

Abstract Background Exposure to toluene diisocyanate (TDI) is a significant pathogenic factor for asthma. We previously reported that the receptor for advanced glycation end products (RAGE) plays a key role in TDI-induced asthma. Histone deacetylase (HDAC) has been reported to be important in asthma...

وصف كامل

التفاصيل البيبلوغرافية

المؤلفون الرئيسيون:	Xianru Peng, Minyu Huang, Wenqu Zhao, Zihan Lan, Xiaohua Wang, Yafei Yuan, Bohou Li, Changhui Yu, Laiyu Liu, Hangming Dong, Shaoxi Cai, Haijin Zhao
التنسيق:	Artigo
اللغة:	Inglês
منشور في:	BMC 2022-02-01
سلاسل:	BMC Pulmonary Medicine
الموضوعات:	Toluene diisocyanate (TDI) Asthma Histone deacetylase 1 (HDAC1) Receptor for advanced glycation end products (RAGE)
الوصول للمادة أونلاين:	https://doi.org/10.1186/s12890-022-01832-3
الوسوم:	إضافة وسم لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!