RAGE mediates airway inflammation via the HDAC1 pathway in a toluene diisocyanate-induced murine asthma model

Abstract Background Exposure to toluene diisocyanate (TDI) is a significant pathogenic factor for asthma. We previously reported that the receptor for advanced glycation end products (RAGE) plays a key role in TDI-induced asthma. Histone deacetylase (HDAC) has been reported to be important in asthma...

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Autors principals:	Xianru Peng, Minyu Huang, Wenqu Zhao, Zihan Lan, Xiaohua Wang, Yafei Yuan, Bohou Li, Changhui Yu, Laiyu Liu, Hangming Dong, Shaoxi Cai, Haijin Zhao
Format:	Artigo
Idioma:	Inglês
Publicat:	BMC 2022-02-01
Col·lecció:	BMC Pulmonary Medicine
Matèries:	Toluene diisocyanate (TDI) Asthma Histone deacetylase 1 (HDAC1) Receptor for advanced glycation end products (RAGE)
Accés en línia:	https://doi.org/10.1186/s12890-022-01832-3
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