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Calciprotein particles disrupt autophagy in vascular endothelial cells and smooth muscle cells

Calciprotein particles (CPPs) act as buffers against mineral overload, but during prolonged mineral stress they convert from the small, amorphous CPP1 form into large, crystalline CPP2. This shift is associated with endothelial dysfunction and arterial stiffness. In this brief report, we assessed th...

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Bibliografische gegevens
Hoofdauteurs: Negar Sharifimoghaddammood, Isabel Pintelon, Winnok H. De Vos, Lynn Roth, Cédric H.G. Neutel
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Elsevier 2026-09-01
Reeks:Atherosclerosis Plus
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Online toegang:http://www.sciencedirect.com/science/article/pii/S266708952600012X
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