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Calciprotein particles disrupt autophagy in vascular endothelial cells and smooth muscle cells

Calciprotein particles (CPPs) act as buffers against mineral overload, but during prolonged mineral stress they convert from the small, amorphous CPP1 form into large, crystalline CPP2. This shift is associated with endothelial dysfunction and arterial stiffness. In this brief report, we assessed th...

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Detaylı Bibliyografya
Asıl Yazarlar: Negar Sharifimoghaddammood, Isabel Pintelon, Winnok H. De Vos, Lynn Roth, Cédric H.G. Neutel
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Elsevier 2026-09-01
Seri Bilgileri:Atherosclerosis Plus
Konular:
Online Erişim:http://www.sciencedirect.com/science/article/pii/S266708952600012X
Etiketler: Etiketle
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