NF-κB activation by <it>Helicobacter pylori </it>requires Akt-mediated phosphorylation of p65
<p>Abstract</p> <p>Background</p> <p>The inflammatory response in <it>Helicobacter pylori</it>-infected gastric tissue is mediated by <it>cag </it>pathogenicity island (PAI)-dependent activation of nuclear factor-κB (NF-κB). Phosphatidylinositol 3-kinase (PI3K)/Akt signaling is known to play a role...
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| Hoofdauteurs: | , , , , , , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
BMC
2009-02-01
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| Reeks: | BMC Microbiology |
| Online toegang: | http://www.biomedcentral.com/1471-2180/9/36 |
| Tags: |
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