NF-κB activation by <it>Helicobacter pylori </it>requires Akt-mediated phosphorylation of p65

<p>Abstract</p> <p>Background</p> <p>The inflammatory response in <it>Helicobacter pylori</it>-infected gastric tissue is mediated by <it>cag </it>pathogenicity island (PAI)-dependent activation of nuclear factor-κB (NF-κB). Phosphatidylinositol 3-kinase (PI3K)/Akt signaling is known to play a role...

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Bibliografische Detailangaben

Hauptverfasser:	Tomita Mariko, Sawada Shigeki, Ishikawa Chie, Kawakami Hirochika, Tomimori Koh, Takeshima Eriko, Senba Masachika, Kinjo Fukunori, Mimuro Hitomi, Sasakawa Chihiro, Fujita Jiro, Mori Naoki
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	BMC 2009-02-01
Schriftenreihe:	BMC Microbiology
Online-Zugang:	http://www.biomedcentral.com/1471-2180/9/36
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