IgLON5 deficiency produces behavioral alterations in a knockout mouse model

BackgroundAnti-IgLON5 disease is a neurological disorder characterized by autoantibodies against IgLON5 and pathological evidence of neurodegeneration. IgLON5 is a cell adhesion molecule of unknown function that is highly expressed in the brain. Our aim was to investigate the impact of IgLON5 loss-o...

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Autori principali:	Jon Landa, Ana Beatriz Serafim, Mercedes Alba, Estibaliz Maudes, Laura Molina-Porcel, Anna Garcia-Serra, Francesco Mannara, Josep Dalmau, Francesc Graus, Lidia Sabater
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	Frontiers Media S.A. 2024-02-01
Serie:	Frontiers in Immunology
Soggetti:	Iglon5 animal model knockout behavior (mice) anti-IgLON5 antibody encephalopathy
Accesso online:	https://www.frontiersin.org/articles/10.3389/fimmu.2024.1347948/full
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