Cell‐cycle‐specific lesion evolution rather than inhibition of double‐strand‐break repair underpins cisplatin radiosensitization
DNA double‐strand breaks (DSBs) generated from collisions of DNA replication forks with cisplatin‐induced interstrand crosslinks underpin cisplatin cytotoxicity. Yet, the impact of cell‐cycle‐dependent cisplatin–DNA adduct (CDA) formation on cell cycle progression and interactions with DNA replicati...
Kaydedildi:
| Asıl Yazarlar: | , , , , , , , , , , |
|---|---|
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Wiley
2026-07-01
|
| Seri Bilgileri: | Molecular Oncology |
| Konular: | |
| Online Erişim: | https://doi.org/10.1002/1878-0261.70240 |
| Etiketler: |
Etiket eklenmemiş, İlk siz ekleyin!
|