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Docking and stability defects in mitofusin highlight the proteasome as a potential therapeutic target

Summary: Defects in mitochondrial fusion are at the base of many diseases. Mitofusins power membrane-remodeling events via self-interaction and GTP hydrolysis. However, how exactly mitofusins mediate fusion of the outer membrane is still unclear. Structural studies enable tailored design of mitofusi...

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Principais autores: Ira Buntenbroich, Vincent Anton, Daniel Perez-Hernandez, Tânia Simões, Felix Gaedke, Astrid Schauss, Gunnar Dittmar, Jan Riemer, Mafalda Escobar-Henriques
Format: Artigo
Sprog:Inglês
Udgivet: Elsevier 2023-07-01
Serier:iScience
Fag:
Biological sciences
Genetics
Molecular biology
Cell biology
Online adgang:http://www.sciencedirect.com/science/article/pii/S258900422301091X
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