Differential effects of buffer pH on Ca2+-induced ROS emission with inhibited mitochondrial complex I and III

Excessive mitochondrial reactive oxygen species (ROS) emission is a critical component in the etiolo-gy of ischemic injury. Complex I and complex III of the electron transport chain are considered the primary sources of ROS emission during cardiac ischemia and reperfusion (IR) injury. Several factor...

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Principais autores:	Daniel P Lindsay, Amadou K S Camara, David F Stowe, Ryan eLubbe, Mohammed eAldakkak
Formato:	Artigo
Idioma:	Inglês
Publicado em:	Frontiers Media S.A. 2015-03-01
coleção:	Frontiers in Physiology
Assuntos:	pH Complex I Ca2+ Mitochondrial Permeability Transition Pore reactive oxygen species (ROS) complex III
Acesso em linha:	http://journal.frontiersin.org/Journal/10.3389/fphys.2015.00058/full
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