Prenatal PM2.5 exposure contributes to neuronal tau lesion in male offspring mice through mitochondrial dysfunction-mediated insulin resistance

The epidemiological evidence has linked prenatal exposure to fine particulate matter (PM2.5) pollution with neurological diseases in offspring. However, the biological process and toxicological mechanisms remain unclear. Tau protein is a neuronal microtubule-associated protein expressed in fetal bra...

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Autors principals:	Yanwen Hou, Wei Wei, Guangke Li, Nan Sang
Format:	Artigo
Idioma:	Inglês
Publicat:	Elsevier 2022-11-01
Col·lecció:	Ecotoxicology and Environmental Safety
Matèries:	Prenatal exposure PM2.5 Tau lesion Insulin resistance Mitochondrial dysfunction
Accés en línia:	http://www.sciencedirect.com/science/article/pii/S0147651322009915
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