Autophagic degradation determines the fate of T315I-mutated BCR-ABL protein

Παρόμοια τεκμήρια

• The quantitative level of T315I mutated BCR-ABL predicts for major molecular response to second-line nilotinib or dasatinib treatment in patients with chronic myeloid leukemia
  από: Thoralf Lange, κ.ά.
  Έκδοση: (2013-05-01)
• Autophagic degradation determines the fate of T315I-mutated BCR-ABL protein
  από: Haruka Shinohara, κ.ά.
  Έκδοση: (2019-05-01)
• Computational Insights into the Polypharmacological Landscape of BCR-ABL Inhibitors: Emphasis on Imatinib and Nilotinib
  από: Rima Hajjo, κ.ά.
  Έκδοση: (2025-06-01)
• Quantitative detection of T315I mutations of BCR::ABL1 using digital droplet polymerase chain reaction
  από: Huijun Mu, κ.ά.
  Έκδοση: (2024-12-01)
• Advances in Targeting BCR-ABL^T315I Mutation with Imatinib Derivatives and Hybrid Anti-Leukemic Molecules
  από: Aleksandra Tuzikiewicz, κ.ά.
  Έκδοση: (2026-01-01)