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Alleviation of accelerated diabetic atherogenesis in STZ-treated apoE/NOX1 DKO mice, apoE−/−/tg-EC-DHFR mice, and by folic acid

We and others have previously shown that uncoupling of endothelial nitric oxide synthase (eNOS) induces oxidative stress in diabetes, contributing to endothelial dysfunction. Activation of NADPH oxidase (NOX) isoform NOX1 by angiotensin II (Ang II) triggers eNOS uncoupling via deficiency in dihydrof...

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Principais autores: Yixuan Zhang, Ji Youn Youn, Kai Huang, Yuhan Zhang, Hua Cai
Format: Artigo
Sprog:Inglês
Udgivet: Elsevier 2025-05-01
Serier:Redox Biology
Fag:
Diabetes
Atherosclerosis
Diabetic atherogenesis
Dihydrofolate reductase (DHFR)
Endothelial-specific DHFR transgenic mice (tg-EC-DHFR)
Endothelial nitric oxide synthase (eNOS) uncoupling
Online adgang:http://www.sciencedirect.com/science/article/pii/S2213231725000837
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