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TRIM21 promotes astrocyte-mediated neuroinflammation in experimental autoimmune encephalomyelitis by stabilizing RGMa via K33-linked ubiquitination

Abstract Abnormal astrocytes activation contributes to disease progression in multiple sclerosis (MS), yet the regulatory mechanisms are not fully understood. Here, we found that repulsive guidance molecule a (RGMa) was significantly upregulated in astrocytes in a mouse model of experimental autoimm...

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Principais autores: Shaoru Zhang, Zijie Wang, Ruiqi Cheng, Xiaolin Yang, Anan Jiang, Li Zhu, Xinyue Qin
Format: Artigo
Sprog:Inglês
Udgivet: BMC 2026-03-01
Serier:Journal of Neuroinflammation
Fag:
RGMa
TRIM21
Ubiquitination
Astrocytes
Experimental autoimmune encephalomyelitis.
Online adgang:https://doi.org/10.1186/s12974-026-03769-4
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