From plant extract to molecular panacea: a commentary on Stone (1763) ‘An account of the success of the bark of the willow in the cure of the agues’

Por Wood, John N.
Publicado no Philos Trans R Soc Lond B Biol Sci (2015)

Pain Genes

Por Foulkes, Tom, Wood, John N.
Publicado em 2008

Fat location defines sensation

Por Oh, Uhtaek, Wood, John N.
Publicado em 2009

Worm sensation!

Por Drew, Liam J, Wood, John N
Publicado em 2005

FM1-43 is a permeant blocker of mechanosensitive ion channels in sensory neurons and inhibits behavioural responses to mechanical stimuli

Por Drew, Liam J, Wood, John N
Publicado em 2007

Pain as a channelopathy

Por Raouf, Ramin, Quick, Kathryn, Wood, John N.
Publicado em 2010

Significant Determinants of Mouse Pain Behaviour

Por Minett, Michael S., Eijkelkamp, Niels, Wood, John N.
Publicado em 2014

Mechanical allodynia

Por Lolignier, Stéphane, Eijkelkamp, Niels, Wood, John N.
Publicado no Pflugers Arch (2014)

Kinetic properties of mechanically activated currents in spinal sensory neurons

Por Rugiero, François, Drew, Liam J, Wood, John N
Publicado em 2010

Na(v )1.8-null mice show stimulus-dependent deficits in spinal neuronal activity

Por Matthews, Elizabeth A, Wood, John N, Dickenson, Anthony H
Publicado em 2006

Na(v)1.7 and other voltage-gated sodium channels as drug targets for pain relief

Por Emery, Edward C, Luiz, Ana Paula, Wood, John N
Publicado no Expert Opin Ther Targets (2016)

A sensory neuron-specific, proton-gated ion channel

Por Chen, Chih-Cheng, England, Steven, Akopian, Armen N., Wood, John N.
Publicado em 1998

Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8

Por Nassar, Mohammed A, Levato, Alessandra, Stirling, L Caroline, Wood, John N
Publicado em 2005

Modulation of sensory neuron mechanotransduction by PKC- and nerve growth factor-dependent pathways

Por Di Castro, Amalia, Drew, Liam J., Wood, John N., Cesare, Paolo
Publicado em 2006

Pain channelopathies

Por Cregg, Roman, Momin, Aliakmal, Rugiero, Francois, Wood, John N, Zhao, Jing
Publicado em 2010

The role of Na(v)1.9 channel in the development of neuropathic orofacial pain associated with trigeminal neuralgia

Por Luiz, Ana Paula, Kopach, Olga, Santana-Varela, Sonia, Wood, John N.
Publicado no Mol Pain (2015)

The Tetrodotoxin-Resistant Na(+) Channel Nav1.8 is Essential for the Expression of Spontaneous Activity in Damaged Sensory Axons of Mice

Por Roza, Carolina, Laird, Jennifer MA, Souslova, Veronika, Wood, John N, Cervero, Fernando
Publicado em 2003

GTP up-regulated persistent Na(+) current and enhanced nociceptor excitability require Na(V)1.9

Por Östman, Johan A R, Nassar, Mohammed A, Wood, John N, Baker, Mark D
Publicado em 2008

GTP-induced tetrodotoxin-resistant Na(+) current regulates excitability in mouse and rat small diameter sensory neurones

Por Baker, Mark D, Chandra, Sonia Y, Ding, Yanning, Waxman, Stephen G, Wood, John N
Publicado em 2003

Serum Response Factor Mediates NGF-Dependent Target Innervation by Embryonic DRG Sensory Neurons

Por Wickramasinghe, S. Rasika, Alvania, Rebecca S., Ramanan, Narendrakumar, Wood, John N., Mandai, Kenji, Ginty, David D.
Publicado em 2008