Investigation of the Fate of Type I Angiotensin Receptor after Biased Activation

Por Szakadáti, Gyöngyi, Tóth, András D., Oláh, Ilona, Erdélyi, László Sándor, Balla, Tamas, Várnai, Péter, Hunyady, László, Balla, András
Publicado no Mol Pharmacol (2015)

Heterologous phosphorylation–induced formation of a stability lock permits regulation of inactive receptors by β-arrestins

Por Tóth, András D., Prokop, Susanne, Gyombolai, Pál, Várnai, Péter, Balla, András, Gurevich, Vsevolod V., Hunyady, László, Turu, Gábor
Publicado no J Biol Chem (2018)

Differential manipulation of arrestin-3 binding to basal and agonist-activated G protein-coupled receptors

Por Prokop, Susanne, Perry, Nicole A., Vishnivetskiy, Sergey A., Toth, Andras D., Inoue, Asuka, Milligan, Graeme, Iverson, Tina M., Hunyady, Laszlo, Gurevich, Vsevolod V.
Publicado no Cell Signal (2017)

Inflammation leads through PGE/EP (3) signaling to HDAC5/MEF2‐dependent transcription in cardiac myocytes

Por Tóth, András D, Schell, Richard, Lévay, Magdolna, Vettel, Christiane, Theis, Philipp, Haslinger, Clemens, Alban, Felix, Werhahn, Stefanie, Frischbier, Lina, Krebs‐Haupenthal, Jutta, Thomas, Dominique, Gröne, Hermann‐Josef, Avkiran, Metin, Katus, Hugo A, Wieland, Thomas, Backs, Johannes
Publicado no EMBO Mol Med (2018)