Combined forced oscillation and forced expiration measurements in mice for the assessment of airway hyperresponsiveness

Por Shalaby, Karim H, Gold, Leslie G, Schuessler, Thomas F, Martin, James G, Robichaud, Annette
Publicado em 2010

Inhaled birch pollen extract induces airway hyperresponsiveness via oxidative stress but independently of pollen-intrinsic NADPH oxidase activity, or the Toll-like receptor 4-TRIF...

Por Shalaby, Karim H., Allard-Coutu, Alexandra, O’Sullivan, Michael, Nakada, Emily, Qureshi, Salman T., Day, Brian J., Martin, James G.
Publicado em 2013

Pathogenic Th17 inflammation is sustained in the lungs by conventional dendritic cells and TLR4 signaling

Por Shalaby, Karim H., Lyons-Cohen, Miranda R., Whitehead, Gregory S., Thomas, Seddon Y., Prinz, Immo, Nakano, Hideki, Cook, Donald N.
Publicado no J Allergy Clin Immunol (2017)

Concomitant Exposure to Ovalbumin and Endotoxin Augments Airway Inflammation but Not Airway Hyperresponsiveness in a Murine Model of Asthma

Por Mac Sharry, John, Shalaby, Karim H., Marchica, Cinzia, Farahnak, Soroor, Chieh-Li, Tien, Lapthorne, Susan, Qureshi, Salman T., Shanahan, Fergus, Martin, James G.
Publicado em 2014

TNF is required for TLR ligand–mediated but not protease-mediated allergic airway inflammation

Por Whitehead, Gregory S., Thomas, Seddon Y., Shalaby, Karim H., Nakano, Keiko, Moran, Timothy P., Ward, James M., Flake, Gordon P., Nakano, Hideki, Cook, Donald N.
Publicado no J Clin Invest (2017)

The TLR4–TRIF pathway can protect against the development of experimental allergic asthma

Por Shalaby, Karim H., Al Heialy, Saba, Tsuchiya, Kimitake, Farahnak, Soroor, McGovern, Toby K., Risse, Paul‐Andre, Suh, Woong‐Kyung, Qureshi, Salman T., Martin, James G.
Publicado no Immunology (2017)