Epigenetic mechanisms regulating longevity and aging

por Sen, Payel, Shah, Parisha P., Nativio, Raffaella, Berger, Shelley L.
Publicado no Cell (2016)

Tid1/Rdh54 promotes dissociation of Dmc1 from nonrecombinogenic sites on meiotic chromatin

por Holzen, Teresa M., Shah, Parisha P., Olivares, Heidi A., Bishop, Douglas K.
Publicado em 2006

Swi2/Snf2-related translocases prevent accumulation of toxic Rad51 complexes during mitotic growth

por Shah, Parisha P., Zheng, Xiuzhong, Epshtein, Anastasiya, Carey, Jeffrey N., Bishop, Douglas K., Klein, Hannah L.
Publicado em 2010

H4K44 acetylation facilitates chromatin accessibility during meiosis

por Hu, Jialei, Donahue, Greg, Dorsey, Jean, Govin, Jérôme, Yuan, Zuofei, Garcia, Benjamin A., Shah, Parisha P., Berger, Shelley L.
Publicado no Cell Rep (2015)

A Balance Between Intermediate Filaments and Microtubules Maintains Nuclear Architecture in the Cardiomyocyte

por Heffler, Julie, Shah, Parisha P., Robison, Patrick, Phyo, Sai, Veliz, Kimberly, Uchida, Keita, Bogush, Alexey, Rhoades, Joshua, Jain, Rajan, Prosser, Benjamin L.
Publicado no Circ Res (2019)

Systematic genetic and proteomic screens during gametogenesis identify H2BK34 methylation as an evolutionary conserved meiotic mark

por Crespo, Marion, Luense, Lacey J., Arlotto, Marie, Hu, Jialei, Dorsey, Jean, García-Oliver, Encar, Shah, Parisha P., Pflieger, Delphine, Berger, Shelley L., Govin, Jérôme
Publicado no Epigenetics Chromatin (2020)

Core components of the nuclear pore bind distinct states of chromatin and contribute to Polycomb repression.

por Gozalo, Alejandro, Duke, Ashley, Lan, Yemin, Pascual-Garcia, Pau, Talamas, Jessica A., Nguyen, Son C., Shah, Parisha P., Jain, Rajan, Joyce, Eric F., Capelson, Maya
Publicado no Mol Cell (2019)

Lysosome-mediated processing of chromatin in senescence

por Ivanov, Andre, Pawlikowski, Jeff, Manoharan, Indrani, van Tuyn, John, Nelson, David M., Rai, Taranjit Singh, Shah, Parisha P., Hewitt, Graeme, Korolchuk, Viktor I., Passos, Joao F., Wu, Hong, Berger, Shelley L., Adams, Peter D.
Publicado em 2013

Lamin B1 depletion in senescent cells triggers large-scale changes in gene expression and the chromatin landscape

por Shah, Parisha P., Donahue, Greg, Otte, Gabriel L., Capell, Brian C., Nelson, David M., Cao, Kajia, Aggarwala, Varun, Cruickshanks, Hazel A., Rai, Taranjit Singh, McBryan, Tony, Gregory, Brian D., Adams, Peter D., Berger, Shelley L.
Publicado em 2013

MLL1 is essential for the senescence-associated secretory phenotype

por Capell, Brian C., Drake, Adam M., Zhu, Jiajun, Shah, Parisha P., Dou, Zhixun, Dorsey, Jean, Simola, Daniel F., Donahue, Greg, Sammons, Morgan, Rai, Taranjit Singh, Natale, Christopher, Ridky, Todd W., Adams, Peter D., Berger, Shelley L.
Publicado no Genes Dev (2016)

Senescent cells harbour features of the cancer epigenome

por Cruickshanks, Hazel A., McBryan, Tony, Nelson, David M., VanderKraats, Nathan D., Shah, Parisha P., van Tuyn, John, Rai, Taranjit Singh, Brock, Claire, Donahue, Greg, Dunican, Donncha S., Drotar, Mark E., Meehan, Richard R., Edwards, John R., Berger, Shelley L., Adams, Peter D.
Publicado em 2013

Genome-nuclear lamina interactions regulate cardiac stem cell lineage restriction

por Poleshko, Andrey, Shah, Parisha P., Gupta, Mudit, Babu, Apoorva, Morley, Michael, Manderfield, Lauren J., Ifkovits, Jamie L., Calderon, Damelys, Aghajanian, Haig, Sierra-Pagán, Javier E., Sun, Zheng, Wang, Qiaohong, Li, Li, Dubois, Nicole, Morrisey, Edward E., Lazar, Mitchell A., Smith, Cheryl L., Epstein, Jonathan A., Jain, Rajan
Publicado no Cell (2017)

BRD4 Interacts with GATA4 to Govern Mitochondrial Homeostasis in Adult Cardiomyocytes

por Padmanabhan, Arun, Alexanian, Michael, Linares-Saldana, Ricardo, González-Terán, Bárbara, Andreoletti, Gaia, Huang, Yu, Connolly, Andrew J., Kim, Wonho, Hsu, Austin, Duan, Qiming, Winchester, Sarah A. B., Felix, Franco, Perez-Bermejo, Juan A., Wang, Qiaohong, Li, Li, Shah, Parisha P., Haldar, Saptarsi M., Jain, Rajan, Srivastava, Deepak
Publicado no Circulation (2020)

Autophagy mediates degradation of nuclear lamina

por Dou, Zhixun, Xu, Caiyue, Donahue, Greg, Shimi, Takeshi, Pan, Ji-An, Zhu, Jiajun, Ivanov, Andrejs, Capell, Brian C., Drake, Adam M., Shah, Parisha P., Catanzaro, Joseph M., Ricketts, M. Daniel, Lamark, Trond, Adam, Stephen A., Marmorstein, Ronen, Zong, Wei-Xing, Johansen, Terje, Goldman, Robert D., Adams, Peter D., Berger, Shelley L.
Publicado no Nature (2015)

Pathogenic LMNA variants disrupt cardiac lamina-chromatin interactions and de-repress alternative fate genes

por Shah, Parisha P., Lv, Wenjian, Rhoades, Joshua H., Poleshko, Andrey, Abbey, Deepti, Caporizzo, Matthew A., Linares-Saldana, Ricardo, Heffler, Julie G., Sayed, Nazish, Thomas, Dilip, Wang, Qiaohong, Stanton, Liam J., Bedi, Kenneth, Morley, Michael P., Cappola, Thomas P., Owens, Anjali T., Margulies, Kenneth B., Frank, David B., Wu, Joseph C., Rader, Daniel J., Yang, Wenli, Prosser, Benjamin L., Musunuru, Kiran, Jain, Rajan
Publicado no Cell Stem Cell (2021)