CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease

Por Ladd, Andrea N.
Publicado em 2012

The importance of CELF control: molecular and biological roles of the CUG-BP, Elav-like family of RNA binding proteins

Por Dasgupta, Twishasri, Ladd, Andrea N.
Publicado em 2011

Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy

Por Berger, Dara S., Ladd, Andrea N.
Publicado em 2012

Finding signals that regulate alternative splicing in the post-genomic era

Por Ladd, Andrea N, Cooper, Thomas A
Publicado em 2002

RNA binding proteins in the regulation of heart development

Por Blech-Hermoni, Yotam, Ladd, Andrea N.
Publicado em 2013

Identification of transcripts regulated by CUG-BP, Elav-like family member 1 (CELF1) in primary embryonic cardiomyocytes by RNA-seq

Por Blech-Hermoni, Yotam, Ladd, Andrea N.
Publicado no Genom Data (2015)

Gene Expression Analyses Implicate an Alternative Splicing Program in Regulating Contractile Gene Expression and Serum Response Factor Activity in Mice

Por Dasgupta, Twishasri, Stillwagon, Samantha J., Ladd, Andrea N.
Publicado em 2013

The CELF Family of RNA Binding Proteins Is Implicated in Cell-Specific and Developmentally Regulated Alternative Splicing

Por Ladd, Andrea N., Charlet-B., Nicolas, Cooper, Thomas A.
Publicado em 2001

Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors

Por Fleming, Victoria A, Geng, Cuiyu, Ladd, Andrea N, Lou, Hua
Publicado em 2012

Gene Expression Analyses during Spontaneous Reversal of Cardiomyopathy in Mice with Repressed Nuclear CUG-BP, Elav-Like Family (CELF) Activity in Heart Muscle

Por Dasgupta, Twishasri, Coram, Ryan J., Stillwagon, Samantha J., Ladd, Andrea N.
Publicado no PLoS One (2015)

Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle

Por Blech-Hermoni, Yotam, Dasgupta, Twishasri, Coram, Ryan J., Ladd, Andrea N.
Publicado no PLoS One (2016)

Cardiac Tissue-Specific Repression of CELF Activity Disrupts Alternative Splicing and Causes Cardiomyopathy

Por Ladd, Andrea N., Taffet, George, Hartley, Craig, Kearney, Debra L., Cooper, Thomas A.
Publicado em 2005

The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation

Por Barron, Victoria A., Zhu, Hui, Hinman, Melissa N., Ladd, Andrea N., Lou, Hua
Publicado em 2010

Muscleblind-like 1 is a negative regulator of TGF-β-dependent epithelial-mesenchymal transition of atrioventricular canal endocardial cells

Por Vajda, Natalie A., Brimacombe, Kyle R., LeMasters, Kathryn E., Ladd, Andrea N.
Publicado em 2009

Expression of a Dominant Negative CELF Protein In Vivo Leads to Altered Muscle Organization, Fiber Size, and Subtype

Por Berger, Dara S., Moyer, Michelle, Kliment, Gregory M., van Lunteren, Erik, Ladd, Andrea N.
Publicado em 2011

CUG-BP, Elav-like family member 1 (CELF1) is required for normal myofibrillogenesis, morphogenesis, and contractile function in the embryonic heart

Por Blech-Hermoni, Yotam, Sullivan, Connor B., Jenkins, Michael W., Wessely, Oliver, Ladd, Andrea N.
Publicado no Dev Dyn (2016)

Loss of muscleblind-like 1 promotes invasive mesenchyme formation in endocardial cushions by stimulating autocrine TGFβ3

Por LeMasters, Kathryn E, Blech-Hermoni, Yotam, Stillwagon, Samantha J, Vajda, Natalie A, Ladd, Andrea N
Publicado em 2012

Muscleblind-like 1 is required for normal heart valve development in vivo

Por Coram, Ryan J., Stillwagon, Samantha J., Guggilam, Anuradha, Jenkins, Michael W., Swanson, Maurice S., Ladd, Andrea N.
Publicado no BMC Dev Biol (2015)