Restricted expression of herpes simplex virus lytic genes during establishment of latent infection by thymidine kinase-negative mutant viruses.

Por Kosz-Vnenchak, M, Coen, D M, Knipe, D M
Publicado em 1990

Human glioblastoma cells persistently infected with simian virus 40 carry nondefective episomal viral DNA and acquire the transformed phenotype and numerous chromosomal abnormaliti...

Por Norkin, L C, Steinberg, V I, Kosz-Vnenchak, M
Publicado em 1985

Evidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons.

Por Kosz-Vnenchak, M, Jacobson, J, Coen, D M, Knipe, D M
Publicado em 1993

Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia.

Por Jacobson, J G, Ruffner, K L, Kosz-Vnenchak, M, Hwang, C B, Wobbe, K K, Knipe, D M, Coen, D M
Publicado em 1993

A deletion mutant of the latency-associated transcript of herpes simplex virus type 1 reactivates from the latent state with reduced frequency.

Por Leib, D A, Bogard, C L, Kosz-Vnenchak, M, Hicks, K A, Coen, D M, Knipe, D M, Schaffer, P A
Publicado em 1989

Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.

Por Coen, D M, Kosz-Vnenchak, M, Jacobson, J G, Leib, D A, Bogard, C L, Schaffer, P A, Tyler, K L, Knipe, D M
Publicado em 1989