Interleukin-1 Receptor Signaling Rather than That of Tumor Necrosis Factor Is Critical in Protecting the Host from the Severe Consequences of a Polymicrobe Anaerobic Infection

Eitemau Tebyg

• Interleukin-1 and Tumor Necrosis Factor Receptor Signaling Is Not Required for Bacteria-Induced Osteoclastogenesis and Bone Loss but Is Essential for Protecting the Host from a Mixed Anaerobic Infection
  gan: Chen, Chih-Ping, et al.
  Cyhoeddwyd: (1999)
• Interleukin-1 and Tumor Necrosis Factor Activities Partially Account for Calvarial Bone Resorption Induced by Local Injection of Lipopolysaccharide
  gan: Chiang, Cheng-Yang, et al.
  Cyhoeddwyd: (1999)
• Helicobacter pylori Preferentially Induces Interleukin 12 (IL-12) Rather than IL-6 or IL-10 in Human Dendritic Cells
  gan: Guiney, Donald G., et al.
  Cyhoeddwyd: (2003)
• Nocardia farcinica Activates Human Dendritic Cells and Induces Secretion of Interleukin-23 (IL-23) Rather than IL-12p70
  gan: Eisenblätter, Martin, et al.
  Cyhoeddwyd: (2012)
• Critical Role of the Interleukin-17/Interleukin-17 Receptor Axis in Regulating Host Susceptibility to Respiratory Infection with Chlamydia Species
  gan: Zhou, Xiaohui, et al.
  Cyhoeddwyd: (2009)