Pretreatment of Mice with Clindamycin Improves Survival of Endotoxic Shock by Modulating the Release of Inflammatory Cytokines

Suppression of endotoxin release and subsequent production of inflammatory cytokines is crucial in the treatment of septic shock. We investigated the effect of clindamycin (CLI) on endotoxic shock induced in mice by Escherichia coli lipopolysaccharide (LPS). Mice were treated with CLI (160 to 600 mg...

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Bibliographische Detailangaben
Hauptverfasser: Hirata, Norio, Hiramatsu, Kazufumi, Kishi, Kenji, Yamasaki, Tohru, Ichimiya, Tomoku, Nasu, Masaru
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Society for Microbiology 2001
Schlagworte:
Biologic Response Modifiers
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC90706/
https://ncbi.nlm.nih.gov/pubmed/11502543
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.45.9.2638-2642.2001
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