DNA Repair Protein Rad55 Is a Terminal Substrate of the DNA Damage Checkpoints
Checkpoints, which are integral to the cellular response to DNA damage, coordinate transient cell cycle arrest and the induced expression of DNA repair genes after genotoxic stress. DNA repair ensures cellular survival and genomic stability, utilizing a multipathway network. Here we report evidence...
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| 出版年: | Mol Cell Biol |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Taylor & Francis
2000
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC85806/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10825202/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.20.12.4393-4404.2000 |
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