BCL-2 Is Phosphorylated and Inactivated by an ASK1/Jun N-Terminal Protein Kinase Pathway Normally Activated at G(2)/M

Multiple signal transduction pathways are capable of modifying BCL-2 family members to reset susceptibility to apoptosis. We used two-dimensional peptide mapping and sequencing to identify three residues (Ser70, Ser87, and Thr69) within the unstructured loop of BCL-2 that were phosphorylated in resp...

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Pubblicato in:	Mol Cell Biol
Autori principali:	Yamamoto, Kazuhito, Ichijo, Hidenori, Korsmeyer, Stanley J.
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	Taylor & Francis 1999
Soggetti:	Cell Growth and Development
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC84954/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10567572/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.19.12.8469
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