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NAD(+) precursor modulates post-ischemic mitochondrial fragmentation and reactive oxygen species generation via SIRT3 dependent mechanisms
Global cerebral ischemia depletes brain tissue NAD(+), an essential cofactor for mitochondrial and cellular metabolism, leading to bioenergetics failure and cell death. The post-ischemic NAD(+) levels can be replenished by the administration of nicotinamide mononucleotide (NMN), which serves as a pr...
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| 出版年: | Exp Neurol |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8328278/ https://ncbi.nlm.nih.gov/pubmed/31837320 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2019.113144 |
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